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Kurr
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Posted - 05/22/2010 : 09:31:14
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Aluminum Toxicity
The following information was compiled and submitted by Frank Hartman.
"From the earliest days of food regulation, the use of alum (aluminum sulphate) in foods has been condemned. It is universally acknowledged as a poison in all countries. If the Bureau of Chemistry had been permitted to enforce the law ... no food product in the country would have any trace of ... any aluminum or saccarin. No soft drink would contain caffeine or hebromin; no bleached flour would be in interstate commerce. Our food and drugs would be wholly without adulteration ... and the health of our people would be vastly improved and their life greatly extended."
From History of crime against the Food Laws (1929) by Dr. Wiley, the prime mover behind the original Pure Food Law and Director of the FDA. He resigned in disgust in 1912 over exceptions granted to the law and lack of enforcement.
Aluminum has been exempted from tesitng for safety by the FDA under a convoluted logic wherein it is classified as GRAS. (Generally Regarded As Safe.) It has never been tested by the FDA on its safety and there are NO restrictions whatever on the amount or use of aluminum.
There are over 2000 references in the National Library of Medicine on adverse effects of alumium. The following were extracted to provide a small sample of the range of toxicity of aluminum.
Chemical Registry
Aluminum toxicity has been recognized in many settings where exposure is heavy or prolonged, where renal function is limited, or where apreviously accumulated bone burden is released in stress or illness. Toxicity may include: encephalopathy (stuttering, gait disturbance, myoclonic jerks, seizures, coma, abnormal EEG) osteomalacia or aplastic bone disease ( associated with painful spontaneous fractures, hypercalcemia, tumorous calcinosis ) proximal myopathy, increased risk of infection, increased left ventricular mass and decreased myocardial function microcytic anemia with very high levels, sudden death.
Aluminum is ubiquitous in our environment; it is the third most prevalent element in the earth's crust. The gastrointestinal tract is relatively impervious to aluminum, absorption normally being only about 2%. Aluminum is absorbed by a mechanism related to that for calcium. Gastric acidity and oral citrate favors absorption, and H2-blockers reduce absorption. As is true for several trace elements, transferrin is the primary protein binder and carrier for aluminum in the plasma, where 80% is protein bound and 20% is free or complexed to small molecules such as citrate.
Cells appear to take up aluminum from transferrin rather than from citrate. Purified preparations of ferritin from brain and liver have been found to contain aluminum.
It is not known if ferritin has a specific binding site for aluminum. Factors regulating the migration of aluminum across the blood–brain barrier are not well understood.
Serum aluminum correlates with encephalopathy; red cell aluminum correlates with microcytic anemia, and bone aluminum correlates with aluminum bone disease.
Basal PTH when elevated appears to protect bone and thereby favor CNS toxicity.
Other factors favoring one form of toxicity over another are not well understood.
Aluminum toxicity has been reported to impair the formation and release of parathyroid hormone. The parathyroid glands concentrate aluminum above levels in surrounding tissues. Treatment of aluminum toxicity in renal failure patients often reactivates hyperparathyroidism, which to a certain extent is helpful for bone remodeling and healing.
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Aluminum neurotoxicity in preterm infants receiving intravenous-feeding solutions.
Bishop N.J. – Morley R. – Day J.P. – Lucas A.
From: N Engl J Med (1997 May 29) 336(22):1557-61
Aluminum, a contaminant of commercial intravenous–feeding solutions, is potentially neurotoxic. We investigated the effect of perinatal exposure to intravenous aluminum on the neurologic development of infants born prematurely.
RESULTS: The 90 infants who received the standard feeding solutions had a mean (± SD) Bayley Mental Development Index of 95 ±22, as compared with 98 ±20 for the 92 infants who received the aluminum-depleted solutions (P=0.39). The former were significantly more likely (39 percent, vs. 17 percent of the latter group; P=0.03) to have a Mental Development Index of less than 85, increasing their risk of subsequent educational problems. For all 157 infants without neuromotor impairment, increasing aluminum exposure was associated with a reduction in the Mental Development Index (P=0.03), with an adjusted loss of one point per day of intravenous feeding for infants receiving the standard solutions. In preterm infants, prolonged intravenous feeding with solutions containing aluminum is associated with impaired neurologic development.
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Aluminum-containing emboli in infants treated with extracorporeal membrane oxygenation.
Vogler C. – Sotelo-Avila C. – Lagunoff D. – Braun P. – Schreifels J.A. – Weber T.
From: N Engl J Med (1988 Jul 14) 319(2):75-9
We found fibrin thrombi or thromboemboli at autopsy in 22 of 23 infants with respiratory failure who had been treated with venoarterial extracorporeal membrane oxygenation (ECMO). In addition, distinctive basophilic aluminum-containing emboli were found in 12 of the infants; the distribution of these emboli was similar to that of the thromboemboli, except that an aluminum-containing embolus was found in a lung in only 1 infant. Sixteen infants had pulmonary thrombi or thromboemboli. We also found friable aluminum-containing concretions adhering loosely to the mixing rods of heat exchangers that had been used to warm the blood flowing through the ECMO circuit; such concretions were not present on unused mixing rods. We propose that these aluminum-containing concretions developed as the silicone coating of the heat exchanger wore away and aluminum metal was exposed to warm, oxygenated blood and that fragments of aluminum-containing concretions formed emboli. This hypothesis is supported by the fact that aluminum-containing emboli were generally not present in the lungs, which are bypassed by ECMO.
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Aluminum in parenteral solutions revisited — again.
Klein G.L.
From: Am J Clin Nutr (1995 Mar) 61(3):449-56
It has been a dozen years since aluminum was first shown to contaminate parenteral nutrition solutions and to be a contributing factor in the pathogenesis of metabolic bone disease in parenteral nutrition patients as well as in uremic patients. However, there are no regulations in place to effectively reduce aluminum contamination of various parenterally administered nutrients, drugs, and biologic products. The purpose of this review is fourfold: 1.) to summarize our knowledge of the adverse effects of aluminum on bone formation and mineralization in parenteral nutrition patients; 2.) to discuss the possible role of aluminum in the osteopenic bone disease of preterm infants; 3.) to show how lack of regulations covering aluminum content of parenteral solutions can lead to vulnerability of new groups of patients to aluminum toxicity, the example being given here is that of burn patients
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Aluminum-induced anemia.
From: Am J Kidney Dis (1985 Nov) 6(5):348-52
... many questions still remain unanswered, it is clear that aluminum causes a microcytic hypoproliferative anemia and is a factor responsible for worsening anemia in patients with end-stage renal disease.
Arch Dermatol (1984 Oct) 120(10):1318-22
Three patients had subcutaneous nodules at the sites of previous injections of vaccine containing tetanus toxoid, showed aluminum crystals in the nodules from two patients. From the evidence available, we believe that these nodules are a complication of inoculations with aluminum-containing vaccines.
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Trace metals and degenerative diseases of the skeleton.
Savory J. – Bertholf R.L. – Wills M.R.
From: Acta Pharmacol Toxicol (Copenh) (1986) 59 Suppl 7:282-8
Aluminum related osteodystrophy is the most important manifestation of trace metal toxicity related to degenerative diseases of the skeleton.
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Aspects of aluminum toxicity.
Hewitt C.D. – Savory J. – Wills M.R.
From: Clin Lab Med (1990 Jun) 10(2):403-22
Attention was first drawn to the potential role of aluminum as a toxic metal over 50 years ago, but was dismissed as a toxic agent as recently as 15 years ago. The accumulation of aluminum, in some patients with chronic renal failure, is associated with the development of toxic phenomena; dialysis encephalopathy, osteomalacic dialysis osteodystrophy, and an anemia. Aluminum accumulation also occurs in patients who are not on dialysis, predominantly infants and children with immature or impaired renal function. Aluminum has also been implicated as a toxic agent in the etiology of Alzheimer's disease, Guamiam amyotrophic lateral sclerosis, and parkinsonism-dementia.
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Soft tissue sarcoma associated with aluminum oxide ceramic total hip arthroplasty. A case report.
Ryu R.K. – Bovill E.G. Jr – Skinner H.B. – Murray W.R.
From: Clin Orthop (1987 Mar)(216):207-12
Malignant tumors around fracture fixation implants have been reported sporadically for many years. Recently, however, reports of sarcomatous degeneration around a standard cemented hip arthroplasty and around cobalt-chromium-bearing hip arthroplasties raise new questions of the malignant potential of metallic ends prostheses. Sarcomatous changes around aluminum oxide ceramics seem not to have been reported in the literature. The present report may be the first documented case of an aggressive soft tissue sarcoma detected 15 months after the patient had an uncemented ceramic total hip arthroplasty. If a causal relationship exists, the incidence of this phenomenon in the United States is 250 times greater than would be expected from statistics on soft tissue sarcoma at the hip.
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The role of aluminium for adverse reactions and immunogenicity of diphtheria-tetanus booster vaccine.
Mark A. – Granstrom M.
From: Acta Paediatr (1994 Feb) 83(2):159-63
235 schoolchildren aged 10 years received either a regular, aluminium-adsorbed diphtheria-tetanus vaccine or the same vaccine in fluid form, in order to investigate if local side effects could be diminished by exclusion of aluminium. System reactions were rare and local reactions frequent in both groups but larger local reactions were even more pronounced in the non-adsorbed vaccine group.
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Neurotoxic effects of aluminium on embryonic chick brain cultures.
From: Acta Neuropathol (Berl) (1994) 88(4):359-66
Toxic damage of brain cells by aluminium (Al) is discussed as a possible factor in the development of neurodegenerative disorders in humans. Effects of Al on cell viability (lysosomal and mitochondrial activity) and differentiation (synthesis of cell-specific proteins) were found to the brain area specific with the highest sensitivity observed in optic tectum.
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Aluminium in tooth pastes and Alzheimer's disease.
Verbeeck R.M. – Driessens F.C. – Rotgans J.
From: Acta Stomatol Belg (1990 Jun) 87(2):141-4
The role of aluminium from tooth pastes may be even more important than that from the drinking water.
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Behavioural effects of gestational exposure to aluminium.
Rankin J. – Sedowofia K. – Clayton R. – Manning A.
From: Ann Ist Super Sanita (1993) 29(1):147-52
The involvement of aluminium in the aetiology of a number of human pathological diseases has altered its status from being a nontoxic, nonabsorbable, harmless element. This maybe of particular concern to the developing foetus which is more susceptible to agents and at lower levels than the adult. Little attention has been given to aluminium's potential reproductive toxicity until recently and further research is required for a full evaluation of its toxicity. Our preliminary results demonstrate behavioural and neurochemical alterations in the offspring of mice exposed to aluminium during gestation. Further, the effects of such exposure are also present in the adult animal suggesting persistent changes in behaviour following prenatal exp
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Aluminum, a neurotoxin which affects diverse metabolic reactions.
Joshi J.G.
From: Biofactors (1990 Jul) 2(3):163-9
Experimental evidence is summarized to support the hypothesis that chronic exposure to low levels of aluminum may lead to neurological disorders.
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Distribution of aluminum in different brain regions and body organs of rat.
Vasishta R.K. – Gill K.D.
From: Biol Trace Elem Res (1996 May) 52(2):181-92
In the present study, an attempt has been made to investigate the distribution of aluminum in different regions of brain and body organs of male albino rats, following subacute and acute aluminum exposure. Aluminum was observed to accumulate in all regions of the brain with maximum accumulation in the hippocampus. Aluminum was also seen to compartmentalize in almost all the tissues of the body to varying extents, and the highest accumulation was in the spleen.
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Chronic aluminum-induced motor neuron degeneration: clinical, neuropathological and molecular biological aspects.
Strong M.J. – Garruto R.M.
From: Can J Neurol Sci (1991 Aug) 18(3 Suppl):428-31
Aluminum chloride induces aggregates of phosphorylated neurofilament that mimics the intraneuronal inclusions of amyotrophic lateral sclerosis.
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Estimates of dietary exposure to aluminium.
Pennington J.A. – Schoen S.A.
From: Food Addit Contam (1995 Jan-Feb) 12(1):119-28
Daily intakes of aluminium were estimated for 14 age-sex groups based on the Food and Drug Administration's (FDA) Total Diet Study dietary exposure model. Estimates of aluminium intakes ranged from 0.7 mg/day for 6-11-month-old infants to 11.5 mg/day for 14-16-year-old males. Average intakes for adult men and women were 8-9 and 7 mg/day, respectively. The major contributors to daily intake of aluminium were foods with aluminium-containing food additives, e.g. grain products and processed cheese.
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Aluminum concentrations in tissues of rats: effect of soft drink packaging.
Kandiah J. – Kies C.
From: Biometals (1994 Jan) 7(1):57-60
Canned soft drink fed rats had significantly higher blood, liver and bone aluminum concentration than rats that were given glass bottled soft drink.
Sources of Aluminum
Over the Counter; Deoderants, vaginal douches, baby wipes, skin creams, suntan lotions, toothpaste, buffered asprin, some haemorrhoid and diarrhea products.
Medical; Vaccinations, allergy testing, intervenous solutions, allergens, wound and antacid irrigation, ulcer treatment, blood oxygenization, bone or joint replacement and burn treatment.
Foods; Aluminum cans, foils, containers, baking powder, cake mixes, frozen dough, pancake mixes, self-rising flour, grains, processed cheese.
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From: History of crime against the Food Laws (1929)
by Dr. Riley, the prime mover behind the original Pure Food Law and Director of the FDA. He resigned in disgust in 1912 over exceptions granted to the law and lack of enforcement.
Aluminum has been exempted from testing for safety by the FDA under a convoluted logic wherein it is classified as GRAS. (Generally Regarded As Safe.) It has never been tested by the FDA on its safety and there are NO restrictions whatever on the amount or use of aluminum.
Diseases Associated with Aluminium Intoxication
H. Tomlinson, M.B., Ch.B., MRCS., LRCP
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The silver [is] mine, and the gold [is] mine, saith the LORD of hosts. Hag 2:8 [/b]
He created it. He controls it. He gave it to us for His use. Why did we turn from sound scriptural currency that PROTECTS us?
KJV Bible w/ Strong's Concordance: http://www.blueletterbible.org/
The book of The Hundreds: http://www.land.netonecom.net/tlp/ref/boh/bookOfTheHundreds_v4.1.pdf
The Two Republics: http://www.whitehorsemedia.com/docs/THE_TWO_REPUBLICS.pdf
Good reading: http://ecclesia.org/truth/government.html
A number of people are educated beyond, sometimes way beyond, their intelligence. - Tenbears
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Edited by - Kurr on 05/22/2010 09:35:45 |
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Chemtrails & Monsanto’s Aluminum Resistance gene
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Posted - 05/21/2010 : 11:50:18
